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In the mouse hepatitis virus (MHV)-infected mice, depletion of the CD4 T cells resulted in the marked increase in hepatitis virus titers in the liver and spleen. The CD4 T cells were shown to confer an ability to protect mice from MHV infection even in the presence of CD8 T cells, since restoration of the CD4 T cells could restore the ability of protection. The CD4 T cells preferentially accumulated in the peritoneal cavity over that of the liver and spleen. The ability of recovery was dependent on both CD4 T cell number and function; in CD4-depleted mice, a single injection of CD4 T cells restored the ability to protect from MHV infection. Recent studies have implicated a role for the TGF-beta in the induction of CD4 T cell-dependent immunity. To see if TGF-beta was important for
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